cadmium poisoning

Intervention group mice lymphocytes injury results were lower than the cadmium poisoning group ( P0.05 ) .

干预组小鼠淋巴细胞损伤结果均低于镉中毒组（P0.05）。

But so far , no effective therapy for chronic cadmium poisoning .

但到目前为止，慢性镉中毒尚无有效的治疗方法。

Changes in Trace Elements Contents of Renal Cells in Cadmium Poisoning

镉中毒时肾细胞内微量元素含量的改变

Significance of Urinary Low Molecular Protein Determination in Diagnosing Chronic Occupational Cadmium Poisoning

尿液低分子量蛋白测定在慢性职业性镉中毒诊断中的意义及方法评定

Discussion on an Early Diagnostic Indicator of Renal Damage in Chronic Cadmium Poisoning

慢性镉中毒早期肾损害指标的探讨

Research on Acute Cadmium poisoning in Rabbits

兔急性镉中毒的研究

Pathologic survey on lung impairment induced by cadmium poisoning

镉中毒致肺损伤的病理形态学观察

DNA oxidizing damage and activity of antioxidative enzymes in rats with chronic cadmium poisoning

大鼠DNA氧化损伤及抗氧化酶活力变化与慢性镉中毒

CONCLUSION : Cadmium poisoning can result in lipid peroxidation and DNA oxidative damage in rats .

结论：镉中毒可导致大鼠脂质过氧化和DNA氧化损伤。

Effect of oxidative stress on apoptosis induced by subchronic cadmium poisoning in liver and kidney

氧化应激在亚慢性镉中毒引起的肝肾细胞凋亡中的作用

Objective To understand the possible role of trace elements in renal damage caused by cadmium poisoning and its mechanism .

目的了解微量元素在镉中毒性肾损伤中的作用及机制。

Orthogonal Experimental Analysis of the Renovating Effect of Three Categories of Plant-metallothionein on Cadmium Poisoning Mice

植物金属硫蛋白提取液对镉中毒小鼠的损伤修复作用

Methods Subacute cadmium poisoning model was prepared with cadmium chloride in rats to induce obvious renal damage .

方法用氯化镉制备大鼠亚急性镉中毒模型，使其发生明显的肾损伤。

Effects of zinc and selenium on metallothionein synthesis and lipid peroxidation in cadmium poisoning mice

锌、硒对镉中毒小鼠金属硫蛋白合成及脂质过氧化损伤的影响

Cadmium poisoning blocks the absorption of calcium in the body , and can lead to severe bone disease .

镉将阻碍身体钙的吸收，这将导致严重的骨科病。

The interaction that extract of plant-metallothionein on cadmium poisoning mice in terms of lipid per oxidation

金属硫蛋白提取液配方对镉中毒小鼠血液脂质过氧化的影响

Study of Pathology by Experimental Cadmium Poisoning on Skeleton in Grass Carp Ctenopharyngodon idella

草鱼实验性镉中毒的骨骼病理学研究

Salt-water organisms are known to be more resistant to cadmium poisoning than freshwater organisms .

大家公认淡水生物比海水生物更容易镉中毒。

The effect of experimental cadmium poisoning on hepatopancreas , kidney , skeleton in Ctenopharyngodon idella

草鱼实验性镉中毒对肝胰脏、肾脏和骨骼的影响

The toxic effect of cadmium poisoning , the plant tolerance against cadmium , cadmium in the transfer mechanism of plant and detoxification mechanisms were reviewed .

对镉毒害的毒害效应、植物对镉的耐受机制以及镉在植物体的转运和解毒机制进行了综述。

Cadmium poisoning causes pulmonary oedema , fulminate hepatitis , testicular injury , nephrotoxicity , immunotoxicity and lethality .

镉中毒会引起肺水肿、急性肝炎、睾丸损伤、肾中毒、免疫毒性甚至致死。

Model hens of cadmium poisoning were duplicated by adding different levels of cadmium in the ration to study the toxicity effect and mechanism of cadmium on reproduction in chicken .

本试验以育成母鸡为试验动物，在饲料中添加不同浓度的镉，复制鸡亚慢性镉中毒模型。

Objective To investigate the health status of cadmium-exposed population in a Lead-Zinc mine and its cadmium-polluted area so as to provide infomation for discrimination of chronic cadmium poisoning caused by environmental pollution .

目的了解某铅锌矿及周围镉污染区镉接触者的健康状况，为镉污染所致慢性镉中毒症的判定提供依据。

Conclusion : The interactions existed in the 3 categories of plant metallothionein on cadmium poisoning . There was statistical significance of interaction between category 1 ( mushrooms ) and Category 2 ( fruits ) .

结论：3类植物金属硫蛋白间存在交互作用，其中以第1类（蘑菇类）和第2类（果实类）交互作用有统计学意义。

Objective : The part of plants which was produced in the location of Xinjiang with the high levels of MT was compatibility . we observed the type of the antagonism cadmium poisoning under the high concentration of plant extract on mice MT and its possible mechanism .

目的：对新疆部分MT含量较高的地产植物MT进行配伍组合，观察高含量植物MT提取液对小鼠镉中毒的拮抗作用类型及其可能机制。

The renal proximal tubules damage due to chronic cadmium poisoning usually showed increase in urine alkaline phosphatase ( U-AKP ) activity , urine β 2-microglobulin quantity and urine cadmium quantity . The changes in U-AKP activity were more sensitive than the latter two .

慢性镉中毒性的肾近曲小管损害时，尿中碱性磷酸酶活性及尿β2-微球蛋白、尿镉排泄量增加，前者要比后两项指标更为敏感。

The changes of lysozyme and alkaline phosphatase in urine in cadmium and mercury poisoning renal damage

镉、汞中毒性肾损伤尿中溶菌酶和碱性磷酸酶活性的变化

200 Grass carps with ( 10.8 g ± 1.2 ) g were made experimental cadmium ( Cd ) poisoning .

用200尾（108±12）g的草鱼作实验性镉中毒实验。

Determination of the cadmium content and the function of main organs when acute cadmium poisoning in rats

急性镉中毒重要器官功能与镉含量测定

Objective : The dose effect relationship was studied in cadmium exposure workers to find sensitive and distinguishing diagnostic indicator of cadmium poisoning .

目的：探讨镉接触剂量与肾损害指标之间的剂量－反应关系，寻找较敏感和稳定的镉损害的早期诊断指标。